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For example, H 2 has been demonstrated to reduce the expression of several pro-inflammatory mediators and markers of oxidative stress and apoptosis including TNF-α, IL-6, IL-1β, IL-10, IL-12, chemokine ligand 2 (CCL2), intercellular adhesion molecule 1, NF-κB, nuclear factor of activated T-cells (NFAT), high mobility group box 1 protein, prostaglandin E2, cyclooxygenase-2 (COX2), serum diamine oxidase, tissue MDA, protein carbonyl, TBARs, myeloperoxidase activity, JNK, and caspase-3 bringing there levels within or preventing their levels from diverging away from normal the homeostatic range [161,162,163]

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